L Melville
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The Crespi-Badcock Hypothesis
[The Crespi-Badcock Hypothesis] that autism and schizophrenia are exactly opposite neurological divergences, which develop epigenetically in the womb in response to entirely opposite environmental stressors â a world of excess and a world of scarcity, respectively â and therefore are never comorbid in the same individual. My extrapolation of Crespi-Badcock is that autism spectrum disorder is really a first world problem in the truest sense. It is rapidly increasing in the developed world, but not the developing world, because compared to the environment in which humans evolved, today's first world embryos receive a stream of resources and conditions indicating it will want for nothing. If the developing human does not anticipate needing the usually large amount of brain resources devoted to reading other people in order to survive, this frees up these resources for understanding systems. Essentially, a person on the autism spectrum is a person whose developing brain received the consistent message that they can make it on their own without relying on many other people, and that their brainpower is better devoted to understanding lots of different and new systems to great detail, so that they *can* make it on their own without relying on many other people. Schizophrenia, meanwhile, is largely a disease of the developing world, and of urban slums worldwide. The brain that's able to become schizophrenic, meanwhile, receives a consistent message during development that it's entering a world of great scarcity and insecurity, and being attuned to other people and their needs and what they communicate will be absolutely indispensable to their survival. The kickstarting event for the first psychotic break is usually some sort of forceful rejection or other form of psychologically traumatic social interaction, in the late teens or early 20s for men, and 30s for women. Autistic patients miss messages from other people that are indeed there. Schizophrenic patients see messages from other people that aren't actually there. Hyposensitivity and hypersensitivity to social cues, respectively. Source: I am an independent general practice physician with a strong interest in psychiatry / behavioral health, who is himself on the autism spectrum, and attracts largely patients who are on the autism spectrum.
Gut Bacteria Has Evolved To Influence Our Behaviour
Gut bacteria has evolved to influence our behaviour, either directly or indirectly, to crave foods that benefit it. That's why it's hard to "come off" certain foods. Anecdotally, it's hard to give up high sugar, high fat foods, yet if you go for long enough without them, your desire for them drops massively. I believe it's because the gut flora that likes that food dies off and no longer influences your behaviour. It's very, very hard to prove, but it seems self evident that if bacteria even had the slightest opportunity to evolve a means to do this, it would almost be a certainty.
Low-Pressure Weather Systems Increase The Frequency And Intensity Of Epileptic Seizures
As a former math and physics major, I took meticulous notes of my sonâs epileptic seizures. At one point, I added barometric readings and current weather data to standard information of date, seizure type, and seizure length in seconds. Iâm convinced that low pressure weather systems increased the frequency and intensity of his epileptic seizures. During a trip to North Carolina, the area had an unusually high, stable high pressure system. He didnât have any seizures during our time there. My theory is that high or low pressure weather systems microscopically change the flow of fluids in the brain or other neurologically sensitive areas of the body such as the micro biome of the gut.
Food Addiction Is A Thing
There probably is a food addiction, specifically towards food rich in sugars and fats, but it's still incredibly difficult to prove it, decipher the specific mechanisms, decipher it from eating disorders and establish if it's an addiction to food, an addiction to eatingThis Panda hasn't followed anyone yet
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